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c-Met in Human Malignancy c-Met overexpression, mutation, or aberrant activation occurs in a wide spectrum of human cancers, including: Gastric cancer Lung cancer (particularly non-small-cell lung cancer) Hepatocellular carcinoma Renal cell carcinoma Breast cancer Glioblastoma and other CNS tumours Colorectal cancer In these contexts, c-Met activation promotes tumour growth, metastasis, and drug resistance

Our bodies have a smart way of handling vitamins - they take what they need and ditch the rest

That copper binding is not incidental

Structure and dynamics of a processive Brownian motor: the translating ribosome
Mechanism of M2 macrophage-derived extracellular vesicles carrying lncRNA MEG3 in inflammatory responses in ulcerative colitis

This supplement is typically given within a few days of birth since iron deficiency lowers resistance to illnesses like pneumonia and scours