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Previous studies have shown that the PI3K/AKT signaling pathway plays anti-neuroinflammation, anti-oxidative stress and anti-apoptosis roles in neurons and increases AKT signaling, which inhibits the release of proinflammatory cytokines [42,43,44]

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Investigations for primary (clonal) mast cell disorders should also be performed given their prevalence in MCAS patients [1921]

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A scientific review from Annals of Joint confirms: there is conflicting data on NSAID effects on soft tissue healing, and COX-2 inhibitors are identified as a major factor negatively affecting tendons and ligaments [35]

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