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GSTM1 null has been specifically related to an increase of OS and PFS (Medeiros et al., n = 23) (Dhawan et al., GSTT1 gene, the non-null allele relates to an increase in overall survival and progression free survival (Goekkurt et al., GSTA1 gene showed the T/T genotype (rs3957357) associates with an increase of overall survival (Khrunin et al., GSTM3 gene, the AGG/AGG haplotype (rs1799735) is related to less thrombocytopenia, anemia and neuropathy (Khrunin et al., GSTA1 and GSTM3 genes on cisplatin-based therapy

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Its effects vary by cell type and disease stage: global SIRT1 knockout mice typically develop exacerbated MASLD phenotypes

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The soleus muscle of HU rats and mice becomes atrophic and experiences a slow-to-fast phenotype transition, characterized by an increased expression of the fast myosin heavy chain (MHC) isoform [111, 112]