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Osteoclast-specific inactivation of HIF-1 antagonises osteoporotic bone loss in mice, suggesting that HIF-1 also directly promotes osteoclast activation and bone loss in vivo 19

The high antigen binding was due to a site-directed orientation of the thiol-biotinylated fragments

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Impaired oral hygiene and genetic alterations resulting in susceptibility to malignancies such as Fanconi anemia have also been implicated as risk factors

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CD38 deficiency prevents diabetic nephropathy by inhibiting lipid accumulation and oxidative stress through activation of the SIRT3 pathway