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If the decay extends to the interior of the tooth, including nerves and blood vessels, it can cause an infection

Consistent with this notion, center-to-center distances between SYT1 and PSD-95 nanomodules (either NMDAR or AMPAR colocalized) closely match the distances measured for GluN1 and SYT1 (GluA2 colocalized PSD-95: 347 6.3 nm, p = 0.9967

The delay in clearance of apoptotic cells may prolong interaction between ROS and nuclear debris and generate neo-epitopes that subsequently stimulate broad spectrum of autoantibody formation leading to inflammation and organ damage in SLE [5]

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This process also induces danger signals from glioma cells, promoting the maturation and activation of bone marrow-derived dendritic cells (BMDCs), ultimately reshaping the glioma immune microenvironment and eliciting robust anti-tumor responses