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However, there is a larger pool of data describing the association between plasma cell dyscrasias and vitamin B12 deficiency, and one study found that 13.6% of patients with plasma cell dyscrasias had vitamin B12 deficiency [4]

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thus a deficiency may lead to disruption of DNA synthesis/cell metabolism and have serious clinical consequences

Ma et al, 1996

Nevertheless, as discussed above, an abnormal postmethionine-load homocysteine elevation above fasting levels must be taken into account in the general identification of patients bearing transsulfuration defects, such as vitamin B 6 -deficient subjects or heterozygotes for CBS deficiency

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