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In vitro studies suggest that AX has the potential to protect against liver steatosis by preventing fat accumulation in hepatocytes [866,867], It has been reported that AX may reduce hepatic lipid accumulation by modulating the activity of nuclear receptors PPAR- and PPAR- in a favorable manner, acting as a PPAR- agonist and a PPAR- antagonist, respectively [866]

133 Alternatively, Guo et al

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The study found that the activity of antioxidant enzymes in children with ALL was lower than in controls, and DNA base damage was worse than in controls (Sentrker et al., 1997), supporting the notion of redox imbalance