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PMC Article PMC6073405

The toxins, enzymes and metabolites of bacteria (predominantly Gram-negative anaerobic) present in the dental plaque play a key role in the initiation of the inflammatory process, but the exact pathomechanism is far from being understood in detail (5-8)

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Due to the pathogenic role of C1q and C3 in OL death, axons damage, and myelin loss 49 , we analyzed the C1q and C3 expression in the CC of CPZ mice

42 Exposure to daratumumab also leads to higher levels of granzyme B in T cells, indicating improved killing capacity