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Neuronal genes deregulated in Cornelia de Lange Syndrome respond to removal and re-expression of cohesin

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Abstract The brain is vulnerable to excessive oxidative insults because of its abundant lipid content, high energy requirements, and weak antioxidant capacity

Study of gene-expression changes in dermal fibroblast cultures exposed to GHK-Cu, with focus on collagen, decorin, and matrix-metalloproteinase regulation

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The inverse relationship between TAC and enzyme activity suggests that cows receiving MMMV supplementation experienced lower oxidative stress and thus required less activation of endogenous antioxidant enzymes