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The genetic causes of SCD include homozygosity for the rs334 mutation (HbSS, commonly referred as SCA) and compound heterozygosity between rs334 and mutations that lead to either other structural variants of -globin (such as HbC, causing HbSC) or reduced levels of -globin production as in -thalassemia (causing HbS/-thalassemia)

Excessive ROS damages nucleic acids, proteins, and lipids to induce cell death, and sufficient intracellular GSH is critical for ROS clearance and cellular homeostasis [24,25,26,27,28,29,30]