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In two years old apoE-deficient mice expressing or not endogenous apoC1 [128], apoC1 expression was accompanied by a 70% increase in triglycerides levels and a 30% increase in cholesterol levels which were essentially related to an increase in triglycerides and cholesterol in the VLDL
Interestingly, a live imaging of the fluorescent HCV particle in Huh7.5 cells showed that the particle was first attached on filopodia and then migrated towards the main body of the cell using presumably an actin-dependent transport mechanism[148]
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