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Description

The synthetic version is produced recombinantly for research purposes and mirrors this naturally observed truncated form

Cardiovasc Res 78(2):203212 Weis SM, Cheresh DA (2005) Pathophysiological consequences of VEGF-induced vascular permeability

Intriguingly, this regulatory pathway involves recognition of sialic acids (notably N-acetylneuraminic acid, Neu5Ac) that may be released from injured tissue, implying a feedback mechanism where eosinophils limit their own aggression in response to host damage

Anti-inflammatory Actions: Modulating inflammatory responses, which can be beneficial in research related to chronic inflammation or injury recovery

It is also important to understand that because of the complicated nature of the effects of these treatments, they should only be used under the care of a medical professional with appropriate expertise and experience