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Eliminated mainly in urine, after metabolism by the liver

Thermogravimetric analysis was performed on a TGA8000 (PerkinElmer, USA) instrument from 30 C to 1000 C at a ramping rate of 5 C min 1 in the Ar

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SN excitability activates the NLRP3 vesicles in the heart and generates IL-1 to induce myocardial injury (127), and recent studies have also demonstrated that cardiac insufficiency occurring after MCAO in mice is associated with sustained pro-inflammatory changes in monocytes/macrophages driven by IL-1 (122)