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Using in vitro tag-free mature AIF-based biochemical assays, cellular loss-of-function and gain-of-function studies, and AIF3 splicing models in vivo, we further found that full-length mature AIF was a mammalian mitochondrial complex I alternative NAD(P)H dehydrogenase and possessed previously unrecognized glutathione reductase (GR) activity, whereas AIF3 lost GR and NAD(P)H dehydrogenase activities

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Recently, I-BAR protein MIM was found to contribute to the initiation process

Acetylcholine-induced arteriolar dilation is reduced in streptozotocin-induced diabetic rats with motor nerve dysfunction