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These findings suggest that future therapeutic strategies may focus on enhancing the nerve-bone communication pathway rather than administering CGRP directly

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For example, chemically increasing barrier permeability in young (5 week old) growing mice causes reduced bone density and stunted growth compared to control mice [62]

When the ERK1/2 signaling pathway is activated, it inhibits estrogen production by CYP19 and promotes progesterone generation by STAR (88)