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LPS is a 1020 kDa protein located in the outer membrane of gram-negative bacteria, which is mainly transported across the epithelium via the transcellular route (58)

In contrast to the reduced GAD67 and the consequent attenuation of inhibitory GABAergic neurotransmission, the reduction of GAT-1 mRNA expression tends to increase the synaptic activity of GABA (63)

Saturated FAs represent an endogenous hazards that upregulate the inflammasome in NASH in the form of a first hit and induce sensitization of hepatocytes to a second hit of LPS, resulting in the release of IL-1 [90]