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Autophagy promotes metastasis and glycolysis by upregulating MCT1 expression and Wnt/-catenin signaling pathway activation in hepatocellular carcinoma cells

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Since selenium supplementation in the forms of sodium selenite and SeM was well documented to induce the expressions of seleno-GPxs through their metabolic assimilation into bioavailable selenium (hydrogen selenide), the initial studies focused on exploring selenium supplementation as a strategy to achieve protection against radiation-induced toxicity [38, 66]

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