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MUT MUT (methylmalonyl-CoA mutase) is a mitochondrial enzyme that converts methylmalonyl-CoA (from the earlier synthesised alpha ketobutyrate) to succinyl-CoA with adenosyl-cobalamin as a cofactor

Research to date strongly suggests that interactions among mitochondrial deficits, neurotransmitter dysfunction, oxidative stress, and inflammation produce the symptoms of schizophrenia [140]

Nutrients 5(3):758770

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Phospholipid release has been observed in experimental TBI models, which have shown an acute increase in serum SM and PC species as well as a decrease in serum TGs after mTBI in rats [86]