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These cells enhance the expression of senescence-induced genes such as P16ink4a in B cells, hence expediting systemic aging
Journal of Endocrinology and Metabolism , 4 (3), 64-77
A large body of preclinical evidence suggests that the key role of the IGF axis in cancer is in driving treatment resistance, via general proliferative/survival mechanisms, interactions with other mitogenic signaling networks, and class-specific mechanisms such as DNA damage repair
In a study of drug-induced perturbations in bile acid homeostasis, Yamazaki et al
doi: 10.3748/WJG.V29.I20.3048
