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Purpose To investigate the effect of mitochondrial dysfunction on the autoregulation of blood flow, by measuring levels of glutathione, an indicator of mitochondrial dysfunction, in glaucoma patients

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To provide quantitative data on the possible role of HNE in oxidative genotoxicity, we applied an accurate HPLC-MS approach to the quantification of HNE adducts to DNA and of HNE conjugates to glutathione (HNE-GSH), the product of the major detoxification pathway of HNE in cells

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ALC may also increase dopamine activity and enhance cerebro-vascular and cardio-vascular blood

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One caveat the headlines miss: Category 1 reclassification is not FDA approval

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