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These fibrils may or may not hold clinical significance relative to early aggregates, but the location of early onset point mutations at the dimeric interface provides evidence that these mature structures may be weakened, or at least altered, leading to rapid formation and increased populations of toxic soluble and poorly defined low-n oligomers

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Acknowledgements This work was supported by the Swedish Brain Foundation (grant FO2023-0241), the Swedish Research Council (VR-NT grant 2022-04198, VR-TVAR 2021-03293), and the Science for Life Laboratory (all to P.E

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