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In receptor-deficient systems, the peptide does not produce measurable changes in lipolytic signaling, supporting the conclusion that its effects are mediated through adrenergic pathway engagement rather than direct hormone receptor activation

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however, this does not involve -arrestin2 or NEDD4 (which are critical for agonist-induced ubiquitination), but requires a transmembrane RING-finger E3 ligase, MARCH2 (Membrane-associated RING-CH2) [102]

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The conjugation with the palmitoyl introduces the potential for more consistent delivery across the skin and better stability to skin proteases